Wednesday, June 19, 2013

What are Complications of Acute Glomerulonephritis and Reasons

Acute glomerulonephritis (hereinafter referred to as accelerated nephritis) disease is a group of rapid development, rapid development by proteinuria, hematuria without urinary urine for renal failure. Acute glomerulonephritis has many complications.
Acute glomerulonephritis complications mainly include severe congestive state cycle, hypertensive encephalopathy and acute renal failure. In recent years, with the strengthen of prevention and control work, the incidence and case fatality rate of acute glomerulonephritis has decreased obviously. In the following paragraph, i will introduce the complication of acute glomerulonephritis one by one.
1. Circulation pump: Clinical manifestations are short of breath, chest tightness, cough, lung wet rale, liver tenderness caused by a blood volume expansion, and is different from real cardiac pump failure. Measure the cardiac often increase and not decrease, the cycle time of normal, arteriovenous difference of oxygen partial pressure did not see more, and digitalis kind of cardiotonic effect not beautiful, and the application of the diuretics can often make their ease. A few of intensive development to the real heart failure, rapid emergence from several hours to 1 ~ 2 days after life-threatening pulmonary edema.
2. Hypertension encephalopathy: Hypertension encephalopathy refers to the blood pressure increased sharply and appear central nervous system symptoms. Generally occured to children than adults. Hypertensive encephalopathy often thought to be on the basis of systemic hypertension, small resistance in the brain vascular spasm resulting in lack of brain edema and causes; But some people think that hypertension encephalopathy is a dramatic increase in blood pressure, cerebral original with automatic control function is out of control, cerebrovascular highly congested during contraction, cerebral edema and acute nephritis in addition to water sodium retention may also play a role in the pathogenesis.
3. Acute kidney failure: acute nephritis patients in quite a part in the acute phase have varying degrees of nitrogen qualitative hematic disease, but only a few of them with the progression of acute renal failure. Complications is still lack of effective prevention measures. This has become the leading cause of death in acute nephritis. Clinical manifestations of oliguria or anuria, blood urea nitrogen, serum creatinine increased, high potassium and metabolic acidosis. Oliguria or anuria will continue for 3 ~ 5 or more than 1 week, then urine output gradually increased, symptoms disappeared and the kidney function recovered.
4. Secondary bacterial infections: acute nephritis due to systemic resistance is reduced, Yi Jifa infection. Lung and urinary tract infection are the most common seen. Once occur the secondary infection, the acute glomerulonephritis patients should actively follow the treatment, lest cause the original worse.
Thses are the introduction of the complications of acute glomerulonephritis. So we should pay attention to the acute glomerulonephritis and the complications of acute glomerulonephritis. If you have some other questiona about acute glomerulonephritis and the treratment of acute glomerulonephritis, please consult our online kidney exports. Thank you.

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